![]() Using 3D reconstruction of AA geometries can provide a good approximation of using the same measurement location in a relatively short time. The concurrent increase in hypertension may be the cause and effect of increased arterial stiffness in elderly people. Hypertension patients have a greater pressure-strain modulus than normal people, and they frequently experience hypertension symptoms. The presence of collagen fiber un-crimping and elastin fiber fracture may explain the positive correlation found between age and diameter in this study. AA mechanics appear to go through a transition period during the fifties in most cases, when mechanical properties begin to decline. įor our young group (42.6-5.1 years), there was a mean circumferential strain between diastole and systole of 0.12 0.03, as opposed to 0.046 0.03 for the older group. In the Laplace equation, * = (PR)/t, the stress along the circumference of a cylindrical pressure vessel is expressed as a function of the luminal pressure, the vessel radius, R, and the wall thickness, *. 1g), a closed smooth curve was generated from points around the circumference (Fig. To determine the circumference of the AA for each phase (Fig. The measurements were made at the level of the right pulmonary artery by bisecting the orthogonal geometries from the orthogonal phase to the flow direction of the ascending aorta. Figure 1e depicts a 3D aortic root geometry for a single representative patient, each color representing a single phase of the geometry. The 3D AA and aortic root geometry of each patient was reconstructed to study the effect of age on them. The brachial sphygmomanometer was used to measure both the patient’s age as well as his or her diastolic and systolic blood pressure. CT scanning was performed on a GE LightSpeed 64-channel volume with Avizo (Burlington, MA) software. Thirty-five male patients with suspected coronary artery disease were studied for multiphase cardiac computed tomography imaging. According to O’Rourke and Hashimoto, the AA is currently changing over time, with marked dilation (as well as elongation and stiffening) that is attributed to the non-living components fatigue of the tissue. Prior research on aging of the ascending aorta has been carried out through in vitro imaging studies. According to growing evidence, artery stiffening is a marker and a risk factor for cardiovascular disease. Aging causes vessels to dilate and deteriorate, which increases vessel wall tension, which can lead to aneurysms. Therefore, it is important for people with aortic dilation to be monitored closely by their healthcare provider.ĬT scans were performed on 45 male patients between the ages of 30 and 79 to assess their ascending aorta in vitro characteristics based on their age. However, aortic dilation can also be a sign of a more serious underlying condition, such as aortic aneurysm or aortic dissection. Aortic dilation is a common finding in older adults, and it is generally considered to be a normal part of aging. This dilation is a result of the aortic walls becoming thinner and less elastic. As people age, the aorta begins to dilate, or widen. ![]() The aorta then continues down into the abdomen, where it branches off into the left and right common iliac arteries. The aorta begins at the aortic valve, and it extends upwards into the chest, where it branches off into the left and right common carotid arteries. The aorta is the largest blood vessel in the body, and it is responsible for carrying oxygen-rich blood from the heart to the rest of the body. ![]()
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